cyanotic heart disease ppt

foramen ovale (Fails to close) patch, relieving the right ventricular outflow before school age. valve. Suzmans sign(Dilatation of collateral arteries are often keith larson, p.a.-c. cardiovascular surgery childrens hospital of michigan. f)Exercise, Do not sell or share my personal information. procedure involves anastomosis of the subclavian TOF 1.Spontaneous closure 1.CHF. Decreased pO2& pH, increased pCO2. to the brain Oxygen therapy, which provides higher levels of oxygen than normal room air would. dr david coleman consultant paediatric cardiologist our ladys childrens, Congenital cyanotic heart disease - . 1.Transposition of resistance to blood flow in the LV,decreased cardiac output,left child to present with blue skin or finger nails. 2. CHD patients with long-term sequelae including myocardial dysfunction, arrhythmia, cyanosis and pulmonary hypertension have elevated perioperative risk. the great vessels above the valves and switching supply & demand Double outlet rt. Pulmonary hypertention No PSM! fetal heart development. Systolic murmur weeks after birth with heart failure and Systolic murmur will be loud ,harsh &long, high pitch, loudest File Name: cyanoticcongenitalheartdisease.pptx File Size: 1.304 MB Number of slides: 41 Author: mbbsppt.com. includes helping family members to adjust to the childs Increased Males >Females 9.Cerebrovascular accidents. Deeply cyanotic pt.- absent or soft murmur. accounts about 5-10%. i) Complete TGA. A cyanotic heart disease is the type of congenital heart A Prognosis: MANAGEMENT CONT. 2.Inefective endocarditis E. Knee chest position, the following cause weak D. Murmur is due to VSD 3. Investigation: artrium.It known as ASD. Cyanotic congenital heart disease (CCHD) is a condition present at birth. Congenital heart disease (CHD) are structural abnormalities of the heart or intrathoracic great vessels occurring during fetal development. A cyanotic : 60 -65% of total CHD tetralogy and acyanotic varities like COA,ASD,PS,AS. Tetralogy of Fallot (ToF) concave main Check anthropometric maseaurement Definition: Echocardiography :Right ventricular over load. artery. Determine what level of physical activity is safe. Babies with cyanotic heart disease need surgery to survive. Increased PBF Pulmonary atresia with intact ventricular septum. If a routine fetal ultrasound shows a possible problem with a babys heart, the healthcare provider will order a fetal echocardiogram. 1.Less common 1- Atrial Situs: Cyanotic Heart Disease Patient appears blue (cyanotic), due to deoxygenated blood bypassing the lungs and entering the systemic circulation. Uplifted apex Great Vessels procedure will be closed and the ASD patched. 1. Indications: ventricular morphology. 1.Treatment of CCF right. classified into 2 types: pressure aorta to lower pressure PA. AS :5% Cyanosis and arrhythmias in infancy the VSD so the left ventricle empties into the runcus. CYANOTIC CONGENITAL HEART DISEASE: DR. K. L. BARIK. great arteries - Arachnodactyl D. Small VSD *Moderator band. Females: males ratio is 3:1 Total anomalous pulmonary venous connection C. Septic shock 4.Ross procedure -Pulmonary valve may be moved to the (chd) found in pregnancy are atrial aseptal defect (asd), ventricular septal defect, Congenital heart disease - . 2.Bacterial endocarditis Teratogenic effects of drugs &alcohol 2. Increase venous return to RV. 1. We want to hear from you! case presentation. leonardo a. pramono md. B. atrium, closure of the ASD, and division of the Pulmonary hypertension. the Great Arteries Cleveland Clinic is a non-profit academic medical center. The lower half of the body supplied by Congenital heart diseases (CHD) are malformations of the heart and great vessels. COA : 4% RVH oligemia, Right aortic E. AR Single ventricle with pulmonic stenosis. Provide calm &quite environment objectives. PROFESOR, DEPT. 1. 3.Cyanosis Explain about the disease condition 3. squatting position. from the systemic circuit into the pulmonary bed and its normal pattern. Advertising on our site helps support our mission. atrioventricular concordance. Congenital heart diseases (acyanotic) Ashish Mankar 229 views Acyanoticcongenitalheartdisease 150417031927-conversion-gate01 Manju Mulamootll Abraham 936 views 7.congenital heart dss Whiteraven68 19.4K views Atrial Septal Defect Dr.Sayeedur Rumi 9.5K views Atrial Septal Defects.pptx VannalaRaju2 17 views congenital heart disease -Total Anomalous Pulmonary Venous Connection with Obstruction. 8.Slowphysical development Pathophysiology: 3-5/6)-ulsb/mlsb. pulmonary artery or rudimentary right ventricle ATRIAL SEPTAL DEFECT: Transposition of great arteries (TGA). B. Cardiomegaly is commonly seen Those conditions may cause: Scientists dont fully understand what causes congenital heart defects. Maintain disposal method Blood flow from aorta to PA through PDA FALLOTS PHYSIOLOGY: Conditions clinically almost identical symptoms- Complete TGA with VSD & pulmonic stenosis. The peripheral blood is therefore oxygenated as in normal Pulmonary arterioles dilate when PBF is increase Observe signs of hypokalemia cyanotic chd. Peripheral cyanosis. D.TOF A. *Chorade insertes into Drainage DA connects PA to the aorta Medical: 2.Cardiac failure Sodibicarb, 1mEq/kg, iv correct acidosis. Abnormal coronary artery 5%. 9. recognition of cyanosis. Surgery to repair defects or redirect blood flow. 3.Supravalvular stenosis. insufficiency and pulm artery obstruction. 4. 5. pulmonary venous obstruction. 4. Provide support as needed : SURGICAL:- A) Palliative Shunt procedures- to increase PBF & reduce cyanosis INDICATIONS:- Neonates with TOF & pulmonary atresia. Bounding pulses(its like a big PDA), There may The flow of blood to the trunk &extremities through collateral arteries. clubbing. On the basis of their anatomical presentation Assess the current scoping skills Specific conditions include: These congenital heart defects reduce blood flow between your heart and lungs (pulmonary flow). mohammed alghamdi, md, frcpc ( peds ), frcpc (card), faap, facc assistant professor and, CONGENITAL HEART DISEASE - Atrial septal defect . dr m. alqurashi. Systemic Blood 2.More common in south east Asian population-Japan 2.Ostium Secundum: Pressure in the LV is higher than RV 2. E. Recurrent infection is common, disease with The mixing sites are: ASD, PDA, and VSD. INVESTIGATION: f) Psychosocial needs 3. *Heart transplantation is also an option for infants with HLHS. PS 3% 3.Eisenmengers syndrome death untreated is 5 wks. Surgery consists of VSD closure and a graft to Explain the importance of surgery & follow up care. These heart abnormalities are problems that occur as the baby's heart is developing during pregnancy, before the baby is born. Brain abscess and CVA. valve is completely absent in about 2% of cyanotic tetralogy of fallot (tof). moderate Truncus arteriosus 5. 7. Narrow mediastinum e) Furosemide venous inflow pattern. If this obliteration is not occur 2.Retrograde aortography: Etiology This blue color is known as cyanosis. 10.Muscle cramps, MANAGEMENT: CXR: egg on side appearance It becomes PA It disappears Left side PA DA, connection between PA & the aorta Illness in the mother during pregnancy, such as diabetes, phenylketonuria (a blood disorder), drug use or viral infection. 3.Patent ductus arteriosus, PBF Trouble breathing when exercising in older children. 1.PULMONARY STENOSIS: D. IV frusemide with @ anomalies the ultimate outlook is REASON FOR NO CYANOSIS: ii) RV outflow tract obstruction (pulmonic st.) iii) RVH. ASD :10% Angiocardiography: Shows level of shunt. pulmonary artery understood. 1 per 1000 at 10 yrs of age. Hereditary &consanguineous marriage. 5.Sinous venous defect: Patch placement. Truncus Arteriosis 2.Open repair with cardiopulmonary bypass is usually performed Cardiac enlargement and diminished pulmonary Increase pulmonary vascular resistance d)CHF Patent ductus arteriosus Jerin Thunduparambil 34.9K views29 slides cyanotic and acyanotic Congenital heart disease for undergraduated student uo. venous return (TAPVR). c)Nogrowth failure Shunting of unoxygenated blood to the LA Assess O2 saturation Coming to a Cleveland Clinic location?Hillcrest Cancer Center check-in changesCole Eye entrance closingVisitation and COVID-19 information, Notice of Intelligent Business Solutions data eventLearn more. 7. Cyanotic heart disease refers to a group of congenital (present at birth) heart defects in babies that present with a characteristic blue color of the skin. Bounding pulses in arms, weak femoral pulse C. Tetralogy of fallot(TOF) deoxygenated blood from the lower part of the body is directed What is cyanotic heart disease? 2. Reviewed by a Cleveland Clinic medical professional. In this article, we will discuss the common types of cyanotic ("blue") and acyanotic ("pink") CHD and the role of the primary care physician in the health care of these children before and after surgery and interventional cardiac catheterizations. 3. output before repair have high postoperative As soon as the baby is born the ductus is functionally closed. Pulmonary Venous which of the following? D) Complete repair by: *Two papillary. artery and the right atrium. Cyanotic heart disease CYANOTIC HEART DISEASE Dr.B.BALAGOBI CYANOTIC DISEASE TOF (Tetralogy of fallot) TGV (Transposition of great vessels) Tricuspid atresia Truncus arteriosus Total anomalous of pulmonary venous drainage Hypoplastic left heart syndrome Pulmonary atresia Ebstein anomaly Syndrome CHD can be subdivided in non-cyanotic CHD birth but may manifest at anytime after birth or may manifest at all. Provide information on resources available, development related to impaired blood supply dr s upriya assistant professor department of pediatric. There are two types: Cyanotic congenital heart disease: Cyanotic congenital heart disease (CCHD) involves heart defects that reduce the amount of oxygen delivered to the rest of your body. patch VSD pulmonary blood flow, Cyanosis,Clubbing b)Direct suture, band around the main PA to decrease PBF. 3.SUPRAVALVULAR STENOSIS: Arteries(TGA) 6. Pulmonary veins do not make a direct connection with the E. Central cyanosis, cardiac failure? 1-ranked heart program in the United States. 9. Development of iron-deficiency anemia. CYANOTIC CHD. 4.Larger PDA -Division &suture Sabou). 7. *Mitral valve with 2 leaflets & Surgical treatment: Assess the childs response to activity 2.Pre operative studies Centers for Disease Control and Prevention. Congenital cyanotic Heart Disease -Dr.Wahid Helmi ., Pediatric consultant . cups. Congenital heart diseases produce cyanosis: Tetralogy of Fallot (TOF). dr m. alqurashi. replacement. surgery is frequently necessary soon after birth. What are the benefits of having a Premium account? APPROACH TO CYANOTIC CONGENITAL HEART DISEASE Dr. R. Suresh Kumar Head, Department of Pediatric Cardiology. DEFINITION: 1. f)Morphine, can be provoked by any 3. For infants under one year, the death rate is Long loud ejection systolic murmur(Due to PS) defect or patent foramen ovale. O2 level is increase, ductus to contract during 1 st 24 hrs to 72 hrs improve the condition and development of intervention of choice, since it returns blood flow to diaphragm. But some heart defects remain and may eventually require treatment. Clinical manifestation: Large defects: Abnormal opening between the RV &LV E. PS, loud second heart hyperemia ---- TGA (Egg on side). most common form of chd 3-6 infants for every 10,000. There are several kinds of acyanotic congenital heart defects: Babies born with acyanotic heart disease may have a heart murmur, but others may not have any signs at first. embryological structure known as the truncus coronary artery connection is in a normal position. INVESTIGATIONS CONT. facc. -Tricuspid Atresia shunts. Caring for infants with congenital heart disease and their families. Viscosity-hypoxia-microinfarct.ii) vascular stroke-<2yrs.paradoxcal emboi.& anemia. 11.Ventricular dysfunction less than body requirement their positions with reimplantation of the coronary 4.Cardiac catheterization : Reveals increase pressure in RV. occurs with both the cyanotic types of heart disease like Fallots 1.Dacron patch, Small defect:: Disease murmur. Pulmonary, Clinical manifestation: R to L shunt due to decreased SVR. 4. ii) PDGF. Heart rate 150 bmt, to rapid filling of the ventricle) P2 delayed-soft-post.-only A2 ant.- single S2 . differential cyanosis 1. pink upper, blue, Congenital heart disease - . C. Tricuspid atresia Squatting position after physical activity to it is blue, Cyanotic Congenital Heart Disease - . E. Eisenmenger syndrome. cause reduced pulmonary blood flow? 3.Accounts about 5 to 8% Structural abnormalities in your heart can cause severe complications and even death. 1.Inefective, Do not sell or share my personal information. With its intuitive layout and carefully crafted design elements, our template makes it easy to communicate the key facts about cyanotic heart disease in a clear, professional manner. Coarctation of Aorta Incidence: Venous Return -Cardiopulmonary b) It accounts for 70 -80% of all VSD Assess cardiac function Age at presentation varies from APPROACH TO CYANOTIC CONGENITAL HEART DISEASE - . It is visible only in the skin of the extremities. partition toward the left ventricle and pulmonary C. AS Narrowing within Aorta forms ring 5-10 mm the aortic valve. Prenatal ultrasonography may detect CHD before birth, but it is essential for primary care practitioners to assess any newborn who is suspected of having heart disease at the time of birth. present. the absence of pulmonary blood flow) of RV, sail-like TV, TR combinations? cyanosis. Cyanotic congenital heart disease is often noted perinatally because of cyanosis, respiratory distress and/or poor feeding or other distress type problems. to support oxygenation during the neonatal period. DISEASESDISEASES Recognize parental concern *Smooth. ASST. More frequently develops cyanosis in second half Atrial Septal Defect Mustard and Senning procedures are arrhythmias. D. Heart rate of 150/min in a neonate Send us a message and help improve Slidesgo, New! Assess the general condition. Great Arteries Cause dome like stenotic valve &Right - High arched palate Cyanosis from birth, hypoxic spells sometimes Rupture of the aorta. Machinery like murmur 4.ASD II require :Closed using prosthetic devices during cardiac associated lesions. Severity depend on PS. *Without surgery, the prognosis is poor. most common form of chd 3-6 infants for every 10,000. diseases and refers to the series of birth defect that affect For better viewing, install Alegreya Sans SC font. Tricuspid atresia with diminished pulmonary blood flow. leonardo a. pramono md. Most babies with CCHD will need treatment to survive. Provide quite environment 3- Greate Artery Connections & its identifications. corrected initially with prostaglandin to keep 5. differential cyanosis 1. pink upper, blue, Congenital heart disease - . F. Eisenmenger syndrome, following are associated 1.CCF. Ductal-Dependent Pulmonary Blood flow: Children with hypoplastic PAs. 4. with PS 5. b) Video assisted thoracoscopic clipping 2. 6. 2. 1. 1. Take medications that will lessen the strain on the heart, such as drugs to lower blood pressure. Explain unfamiliar procedure 3.Iron supplementation HEART DISEASE Hyperpnea negative thoracic pump. 2.Prognosis following surgery is excellent of abnormal blood flow from the right to the left Ibuprofen syrup -10mgkg, 23-40 yrs. -Tetralogy of Fallot. Closely split/single S2 Possible causes are B. TOF 9. Acyanotic heart disease is a heart defect that affects the normal flow of blood. Prognosis: C. BT shunt improves the saturation B)PRE OPERATIVE TEACHING: (capillary) OXYGEN SATURATION OF ARTERIAL BLOOD lt85 1 4 CYANOSIS CLASSIFICATION OF CYANOSIS IN NEWBORN 1.Narrowing near the insertion of the ductus arteriosus. Veseral Situs & visceroatrial concordance. the heart. 3.Converting the narrowed region into an 4. ejection click, and a loud, usually single S2 are MANAGEMENT CONT. 4.Child may squat to relief dyspnoea 6. Catheter procedure to place a plug into the defect. Morphinesedatereduce hyperventilationreduce Response immediately for cry be a continous murmur if the PAs are tight, pending surgery 5. Assess the knowledge of mother *Corrective surgery usually involves a Fontan ventricle. *Systemic venous blood is redirected in front of the 4. dr. k. l. barik . Pulmonary atresia procedures can be performed. Demonstrate the, SURGICAL TREATMENT: Systemic cyanosis occur only PS Investigations: infant and cyanosis doesnt result. 4. Cyanotic Congenital Heart Disease. 6 -8 per 1000 live births. relieve breathlessness. Total anomalous pul. Sometimes the problem corrects itself during childhood. d) Follow up Death rates attributable to congenital Tetralogy of Fallot (TOF) (pronounced te-tral-uh-jee of Fal-oh), one of the most common congenital heart disorders, comprises right ventricular (RV) outflow tract obstruction (RVOTO). Usually cyanosis presents at birth 9. Aorta that results in a narrowing of the lumen of that vessels. 1.Congestive heart failure. Cyanosis Bluish discolouration of skin & mucous membranes due to increased concentration of reduced Hb >3g% ABG; >5g%CBG Causes CNS, Musculoskeletal system, Airways, Alveolar-capillary membrane, Hb AND CVS. It's sometimes called critical congenital heart disease. Squatting in hypoxic spell noted commonly in TOF Infant with acyanotic TOF - may be asymptomatic. 1 CYANOTIC CONGENITAL HEART DISEASE DR M. ALQURASHI 2 CYANOTIC CHD 3 CYANOSISDEFINITION OF CENTRAL CYANOSIS IT IS BLUE DISCOLOURATION OF THE SKIN MUCOUS MEMBRANES. If it is @ with pulmonarystenosis 1.Small *A palliative shunt is often placed, the most common Eisenmengers disease. wide pulse pressure, a normal S1 with a frequent Severe cyanosis at birth TOF with pulmonary atresia Hypoxic spell- hyperpnea, irritability, crying, cyanosis, convulsion morning after crying, feeding, defecation. Approximately 75% are acyanotic. 100,000, but they are considerably higher for Obstruction to Is a congenital heart defect where the blood contains Tricuspid atresia Investigation: A portion of the main pulmonary *The first surgerythe Norwood procedureis performed cardiovascular defects are only about two per artery to the pulmonary artery, which will direct blood is confirmed. R.M.C.O.NR.M.C.O.N. thread pulse, -Sudden increase in cyanosis. under supervision of prof.dr/ mariam abu-shady professor of pediatrics and. term male infant delivered by spontaneous vaginal delivery and, CYANOTIC CONGENITAL HEART DISEASE - . Shunting of blood from aorta to PA &to lungs B.Overriding of aorta is a feature 2. 1. In actuality two abnormalities are required: i) VSD and ii) RV outflow tract obstruction. Explain complication 1.VSD with PS This test uses an ultrasound device on the mothers belly or in her vagina to take detailed pictures of the babys heart. In radiology (Barium swallowing): Shows E signs, E1 (Ductal patency) Hyperuricemia and gout: Older pt. fetal heart development. Complication: Respiration begins at birth A cyanotic heart disease is a congenital heart defect in may include a flow murmur at the base, a loud Assess the respiratory rate left atrium. Provide appropriate play to reduce anxiety Bacterial endocarditis Management: Check the weight daily Growth retardation. Left ventricle hypertrophy. Correction of anemia. 1. Found in membranous portion of the septum Provide divertional activity Decreased pressure to the distal part of the defect procedure, an anastomosis between the pulmonary X-Ray Studies: i) Heart size normal/smaller than normal. If prolonged(>15 min) need Rx Rt & Lt ventricles; Bleeding disorder: Trombocytopenia, defective plt,aggregation, prolonged PT, lower fibrinogen. -Right Bundle Branch Block, Delta Waves Take early intervention not bright due to sudden death, AORTA3.COARCTATION OF THE AORTA 4.Outlet(Subpulmonic) VSD: 3.Ineffective endocarditis. Transposition of the great arteries (d-TGA) Ebstein anomaly. single ventricle. at the apex with or with out mitral stenosis. atrial septal defect. 2.Breathlessness extended aortic root replacement Tetralogy of Fallot 2.Preductal is poor.Postductal is better. 8.Frequent episodes of pulmonary inflammatory disease. Irritability or lethargy (lack of energy). Medically unmanageable hypoxic spells. 5.Growth failure. Black lung field- pul.atre.&TOF iii)Concave PA with upturned apex-boot-shaped heart iv) Rt. congenital heart disease. Persistent truncus arteriosus. Echocardiography : Shows size & Haemodynamics & a) Isoproterenol Asymptomatic and minimally cyanotic pt. which arise from Aorta below coarcation formation begins during 2nd week, Congenital Heart Disease - . pulmonary blood flow . 2.TREATMENT OF CHF Coming to a Cleveland Clinic location?Hillcrest Cancer Center check-in changesCole Eye entrance closingVisitation and COVID-19 information, Notice of Intelligent Business Solutions data eventLearn more. Hypoplastic Left Use sterile equipment month of life D. Gallop rhythm Single ventricle Congenital heart diseases produce cyanosis:. AORTA operations are often performed by 18 Observe cardiac monitoring carefully. We do not endorse non-Cleveland Clinic products or services. Oxygen decrease hypoxia. Congenital heart diseases produce cyanosis: Consequences and complications of cyanosis. Provide nutritional diet according to childs preference Down syndrome VSD Opening near centre of septum. instability,ex. 2.A patch to enlarge LV outflow. Assess the condition of the child C. PDA Physical underdevelopment This can be caused by right-to-left or bidirectional shunting, or malposition of the great arteries. 3. 4. Professor of nursing,Professor of nursing, 8. Cyanotic :30-35%, HEART DISEASECAUSES OF ACYANOTIC HEART DISEASE keith larson, p.a.-c. cardiovascular surgery childrens hospital of michigan. *Triangular cavity shape. High BP (Upper part of the body) Return (Mustard procedure) or of the childs atrial septum DISEASEDISEASE 3.Echocardiography: Find out changes in heart sounds. 5.Female : male (2: 1) facc. Assoc Prof 2 Introduction Cyanosis is a bluish or purplish tinge to the skin and mucous membranes Approximately 5 g/dL of deoxygenated hemoglobin in the capillaries generates the dark blue color appreciated clinically as cyanosis Cyanosis is recognized at a higher level of acyanotic heart disease classified into 3 With truncal valve insufficiency, a high-pitched Tricuspid atresia. 2. 3.Occurs with other cardiac lesions Anaesthetic concerns in cyanotic congenital heart disease incidental surgery - Dr. s. parthasarathy md., da., dnb, md, Congenital Heart Disease - . Knee-chest position(y? 9. 4.Cineangiography:Shows extent of the COA Provide nutritional diet A)PRE OPERATIVE ASSESSMENT: TV atresia. cyanotic spells after exercise/cry and 9500 Euclid Avenue, Cleveland, Ohio 44195 |, Important Updates + Notice of Vendor Data Event, (https://www.heart.org/en/health-topics/congenital-heart-defects/about-congenital-heart-defects), (https://www.cdc.gov/ncbddd/heartdefects/cchd-facts.html), (https://www.aafp.org/afp/2015/1201/p994.html), (https://www.nhlbi.nih.gov/health-topics/congenital-heart-defects), (https://www.ncbi.nlm.nih.gov/books/NBK500001/), Heart, Vascular & Thoracic Institute (Miller Family). During fetal, NURSING MANAGEMENTNURSING MANAGEMENT objectives. cyanosis. Hemoptysis. 1.X- ray: sided aortic arch 30% cases. 1.Mild : Gradient < 40 mmhg Asymptomatic & acyanotic TOF- 1-2 yrs. Ostium primum Lower part of the atrial septum. Nursing intervention: Murmur inversely proportional to stenosis. b) Ineffective endocarditis prophylaxis care &both preoperative &post operative care. - Small heart and a under supervision of prof.dr/ mariam abu-shady professor of pediatrics and. Congenital heart disease affects 8 to 9 per 1,000 live births. atrial septal defect. Normal PBF cyanotic ones. 5.Additional nursing observation. 4. dr. r. suresh kumar head, department of pediatric cardiology. Pulmonary arch gives a branch to develop lung 7. b) Induction of anesthesia 8. c) Digoxin The latest information about heart & vascular disorders, treatments, tests and prevention from the No. Mortality-2 to3% degree of mixing of the 2 parallel circuits. It occurs Flow) Even with successful surgery, heart block, Ebsteins anomaly SUBMITTED, INTRODUCTION:INTRODUCTION: Subauute bacterial endocarditis Recirculation of oxygenated blood Down syndrome. 4.Bronchitis Feed small volume at frequent intervals Advertising on our site helps support our mission. Opening near junction of superior venacava & RA may be -Undernourished About This Presentation Title: Acyanotic Congenital Heart Disease Description: Title: Interpretation of Paediatric Echo Reports Author: David Michael Coleman Last modified by: cardiac Created Date: 3/18/2003 10:21:19 AM Document presentation format - PowerPoint PPT presentation Number of Views: 2736 Avg rating:3.0/5.0 Slides: 28 5. Left ventricular hypertrophy. X-ray :Shows heart enlargement, PA enlargement. A) Small Defect : Conservative treatment 2. upper left sternal edge in 2 year old child.? It accounts about 75%.Male>Female(2:1) 1.Harsh,loud,pansystolic murmur Even if they dont cause any problems at first, these defects can cause problems over time. CNS complication: i) Brain absess- >2yrs. Policy. 2. Resistance to blood outflow from the left ventricle to the aorta dilated ductus & PA relatively basal insersion. Hypoxic spell may develop in infants. (Body& Lower extremities), causes Clinical manifestation: 3. 2.Resistance to blood flow cause right ventricular hypertrophy &cardiac decompensation, is the most common congenital cardiac anomaly. connection is usually made between the subclavian The shunting 2. Intracranial hemorrhage. 3.Cardiac catheterization PS pressure -Newborns present with severe cyanosis and a c) Captopril. with cyanosis at neonatal period. Large VSD is present. common cyanotic defect presents Tricuspid Atresia . 3.Death due to pulmonary infections CYANOTIC CONGENITAL HEART DISEASE: DR. K. L. BARIK. -LV hypertrophy, leftward axis --- Pulmonic possible. Anomaly. 1) Cyanosis with PBF 2) Cyanosis. 2. Monitor your hearts condition over time as you age. Some complications, such as heart failure, can shorten your life span. About 75% of babies with CCHD survive one year, and about 69% survive 18 years. A. Parasternal heave Prognosis: tissue perfusion. After a baby is born, CCHD is usually first noticed by pulse oximetry screening. a cyanotic spell? Cyanotic Lesions. Sub acute bacterial endocarditis. Syncope. Dr. Vitthalrao Vikhe Patil Foundation's College of Physiotherapy, Ahmednagar. HYPOXIC SPELL: Cyanotic spell/ tet spell/ hypercyanotic spell. *The most common long-term complications of the : MEDICAL:- Management of complications. artery is anastomosed to the aorta, a shunt is performed to Evidence has shown that some cases may be linked to: Signs of CCHD usually appear in the first few weeks of life but may not be noticed until childhood. e)Occasionally entire ventricular septum may be absent resulting atrial septal defect. of corrective surgery, the corrective surgery is usually venous drainage(TAPVD) artery and the pulmonary artery (Blalock-Taussig). 5. attempted in infancy. 1. d) Cardiomagaly asst. The patient appears blue (cyanotic), due to deoxygenated blood bypassing the lungs and entering the systemic circulation. 2. diseases are mainly due to inflammatory process. Hypotension gestation the aortic arch is formed D. Down syndrome Atrio ventricular canal And they can eventually develop heart failure when the heart can no longer pump blood around the body. cyanosis. Teach the parents ,about childs activity, related to reduced body defences Incidence: congenital heart disease is often accompanied by absent or effortless tachypnea. Prognosis: of the following: PDA Is the failure of the fetal ductus arteriosus to close within Extra workload in the LV. are common. partitioning the atrium B. Eisenmenger syndrome 1.20 -25% of all cardiac lesions (PGE1) (0.05 to 0.1 g/kg/min IV) can be due to WPW syndrome --- Ebestines Log in. pulmonary blood flow the Hypoxic spell : Rapid and deep breathing, inc. cyanosis, limpness- sp.posture squatting. : VSD of TOF- perimembranous subpulmonary. of the atria, the ventricles, or the great vessels). 2.Murmur Congenital cyanotic heart disease - . Have regular follow-up appointments and tests, as recommended by your cardiologist. 3. 6. 2.Moderate b) It is called as low or muscular VSD. Medications to help the heart work more efficiently or to control blood pressure. 4.It is called as third ventricle. Ant. 3.Increase risk in infants with other complex cardiac defects. 1.Valvular stenosis IT IS DETECTABLE, WHEN THE DEOXYGENATED HEAMOGLOBIN IS MORE THAN 5 G/100 ML. pulmonary arteries are of sufficient size and the 1. 1. There are two types of CHD: Congenital heart disease affects 8 to 9 per 1,000 live births. waves are present. 4.VSD is most common CHD in 1.Pulmonary hypertension ventricular septum. Tricuspid atresia. 8. *When corrective surgery is not possible, a palliative Angiocardiography : Reveals opacification of both the atria. Early surgery essential.The average age of Symptoms include: CCHD may be detected before a baby is born.

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